Graves Disease – BCA Version. Based on “Everytime We Touch”. Bergen County Academies Adv Biology Class of 2008 Biology Project consisting of Michelle Chen, George Soussou, & Hardik Jogani. Its a Hardikness Productions!!
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I have been on youtube for about 10 months now, always a journey to adopt a healthy lifestyle and lose weight. With so much going on and falling ill I was not ready to find out that I have hypothyroid. From painful days, anxiety and now where I am… knowing whats wrong and knowing I now need to adapt. Source info: What causes thyroid disease? There are several different causes of thyroid disease. The following conditions cause hypothyroidism: Thyroiditis is an inflammation of the thyroid gland. This can lower the amount of hormones produced. Hashimoto’s thyroiditis is a painless disease of the immune system that is hereditary. Postpartum thyroiditis occurs in 5 percent to 9 percent of women after giving birth. It is usually a temporary condition. Iodine deficiency is a problem affecting approximately 100 million people around the world. Iodine is used by the thyroid to produce hormones. Although prevalent before the 1950s in the USA, iodine deficiency has been virtually wiped out by the use of iodized salt. A non-functioning thyroid gland affects one in 4000 newborns. If the problem isn’t corrected, the child will be physically and mentally retarded. The following conditions cause hyperthyroidism: With Graves’ disease, the entire thyroid gland might be overactive and produce too much hormone. This problem is also called diffuse toxic goiter (enlarged thyroid gland). Nodules might be overactive within the thyroid. A single nodule is called toxic autonomously functioning …
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FDA is notifying healthcare professionals about the risk of serious and possibly fatal liver injury associated with propylthiouracil, an anti-thyroid drug used to treat Graves’ disease. Propylthiouracil is generally considered second-line therapy in Graves’ disease. The drug of choice in most cases is methimazole, because it has been associated with fewer cases of serious liver damage than propylthiouracil. Children are especially vulnerable to liver damage with propylthiouracil. FDA has identified 10 pediatric cases of serious liver injury from propylthiouracil, and none from methimazole. The risk profile is different for patients in the first trimester of pregnancy, however, since methimazole has been associated with embryopathy and propylthiouracil has not. FDA is working to change the propylthiouracil prescribing information to reflect the risk of hepatotoxicity, and the American Thyroid Association plans to update its treatment guidelines for Graves’ disease. In the meantime, FDA is recommending that clinicians reserve the use of propylthiouracil for patients who cannot take methimazole because of allergy or intolerance, or for patients in the first trimester of pregnancy. Propylthiouracil should not be used in children unless they cannot take methimazole and there are no other treatment options available. If propylthiouracil is prescribed, patients should be closely monitored for liver injury, especially during the first six months of treatment. Advise patients to …
Hyperthyroidism in the Form of Graves’ Disease – Understanding Graves’ Disease
What is Graves’ Disease? One of the most common disorders involving the thyroid is Graves’ disease. Since it was first described by the Irish physician Robert James Graves, it has become the leading cause of hyperthyroidism in the world.
Graves’ disease is an autoimmune disease caused by antibodies that mimic the effects of the hormone thyroid-stimulating hormone produced by the pituitary gland. These antibodies cause the thyroid gland to produce more and more thyroid hormone beyond the normal limits. This causes the body’s metabolism to speed up, resulting in the symptoms of hyperthyroidism.
Exactly how and why these antibodies are produced by the body, there is yet little explanation. However, it has been observed that Graves’ disease tends to run in the family so there might be a genetic predisposition to the production of the autoantibodies.
Another theory states that the production of the antibodies could be the result of a bacterial or viral infection as Graves’ disease tends to manifest later in life. One such organism believed to be involved in the development of Graves’ disease is the bacterium Yersinia enterocolitica. There is indirect evidence that there is a structural similarity between the bacterium and the human thyrotropin receptor, making the antibodies produced against Yersinia enterocolitica react with the thyroid gland. There is very little evidence to support this theory and it has been believed that the bacterium is not a direct cause of Graves’ disease but that it only serves to aid in the development of the disease in genetically susceptible individuals.
Spotting Graves’ Disease
Graves’ disease sports all the common signs and symptoms of a hyperthyroid patient but one such stands out against the rest to differentiate Graves’ disease from the rest — exophthalmos.
Exophthalmos is the protrusion of one or both of the eyeballs in most patients afflicted with Graves’ disease. This is due to the edematous swelling of the tissues at the back of the eyeballs and degenerative changes in the extraocular muscles. Immunoglobulins that may react with the eye muscles can be found in the blood of many of these patients. There is also reason to believe that exophthalmos in itself is an autoimmune process as the concentration of these immunoglobulins are highest in patients with increased levels of thyroid-stimulating immunoglobulins.
Patients with exophthalmos may suffer from impaired vision as the protrusion of the eyeball stretches the optical nerve. There may also be the risk of developing corneal irritation and infections because the eyes cannot fully close when the patient sleeps or blinks, causing the drying of the eyes.
Doctors who suspect Graves’ disease in patients may request thyroid blood tests to measure the levels of thyroid hormones and thyroid-stimulating hormone in the blood. Patients afflicted with Graves’ disease mostly have high levels of thyroid hormone and low levels of TSH. This is because the pituitary gland stops its secretion of TSH when the body’s levels of thyroid hormone go beyond normal limits.
Treatment of Graves’ Disease
Patients with Graves’ disease are usually treated with antithyroid drugs like propylthiouracil, methimazole, and carbimazole. Patients may also be treated with radioactive iodine to destroy the hypersecreting thyroid cells. In some cases, surgery may be necessitated although proper preparations may be made as surgical removal of the thyroid gland in hyperthyroid patients is dangerous.
Graves’ disease patients who must undergo surgical removal of the thyroid gland must first undergo several months of treatment with propylthiouracil until they reach normal basal metabolic rates, followed by large doses of iodine one to two weeks immediately before the operation to decrease the swelling and diminish the blood supply of the thyroid. Prior to the development of these preoperative procedures, mortality rate in thyroidectomy patients was rather high but now, it has been reduced to as low as 1 in every 1000 operations.
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Thyrosoothe Overactive Thyroid 50 Ml Native Remedies
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Hyperthyroidism is a medical condition in which the thyroid gland produces too much thyroid hormone (called T3 and T4). As the thyroid gland plays a crucial role in regulating metabolism and has effects on nearly all organs in the body, an overactive thyroid can have far reaching effects on health and body functioning. The most common cause of an overactive thyroid is Graves Disease. Graves Disease is an auto-immune illness in which the immune system begins to attack parts of ones own body – in
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Living with graves disease – what is the best approach?
Living with graves disease can be extremely difficult, and you can actually have a very small chance of leading a normal and happy life, unless you stop the disease. You may be pleased to learn, that there is a new cure available, which has already cured thousands of patients. It is a work of a researcher, who has years of experience with graves disease patients. You can find it here.
The whole approach that is currently used by modern medicine, to treat graves disease is wrong from the get go. Medicine is trying to ‘treat’ the symptoms of the disease, and to replace the natural function of the thyroid, with artificial hormone pills, or other such substitutes. In the short term, that does help, but in the long run in will do you more harm than good. For one, the drugs which are currently used to treat this disease, can damage your liver and heart. Also, your body starts developing a tolerance for the drugs, and if you stop taking them, the symptoms come back stronger. Of course that way of treating the disease works – but it is definitely not the best option – and it certainly will not make living with graves disease comfortable.
Living with the disease can cut off almost any possibility of you, ever leading a normal and happy life, regardless of how good the other circumstances of your life are. That’s why you need to start thinking outside of the box, when it comes to graves disease, and look into alternative, and working natural cures, like the one mentioned in the beginning of the article. Also, it is important to stop using treatments, which doctors recommend, just because everyone else is doing it. You have to think for yourself. The current results of the medical treatment are clear – all patients are living with graves disease, without getting cured, and they are taking expensive, and harmful medication, in order to manage their symptoms.
Wishing you great health!
Nancy shares what procedures are available for Graves’ disease. For more on thyroid conditions visit www.empowher.com
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